[Homesteadingfamily] Re: Cookware for prepping

My mom once said that if you ate nothing but macaroni and cheese you would probably get cancer because too much of anything is wrong.

Now while I can understand that it could be detrimental to overload your system with iron, I have always had difficulty absorbing iron and have lived most of my life borderline anemic. Here is a good rule of thumb. If your blood is the color of dark red garnets or darker you do not have your iron too low. Ruby colored blood or lighter is anemic.

My aunt who has always had the ability to dissolve the back of her watches in 2 months has blood the color of dark chocolate. She now has problems with Cellulitis. Your info explains some of her difficulty with this disease.

Too little iron and you body can't perform necessary chemical reactions to keep your body functioning properly. It acts as a catelyst. This reduces the temperature that the other chemicals need to join together from about 400 degrees to around 120 degrees which is the core temp of the human body. The iron itself is unchanged.

Kate Kestner

--- In Homesteadingfamily@yahoogroups.com, Dave and Paula Spalding <daveandpaula0923@...> wrote:
>
> Sorry Eric. and thank you for the information. I am very weary of "medical research" and the papers they publish - they are only as good as the subjects you get and the honesty of the doctors who want to publish. Post-menopausal women are extremely biologically different, especially age and hormones. I have always believed that age and lifestyle have a much bigger impact on things like heart attacks and of course, family history.
>
> I just don't believe the small amount of iron that might leach from use of an iron skillet is going to impact someone's health.
>
> So for now, I guess we will just have to agree to disagree. I for one don't use many iron utensils, but that is because I don't like the way they cook most foods and they are way more high maintenance than my non-stick and I am fairly lazy in the 'cleaning-up-after-myself" arena!
>
> --- On Mon, 11/15/10, Eric <local_maxima@...> wrote:
>
>
> From: Eric <local_maxima@...>
> Subject: Re: Cookware for prepping
> To: "Dave and Paula Spalding" <daveandpaula0923@...>
> Date: Monday, November 15, 2010, 8:46 PM
>
>
> Here's a copy of the message I referred to. With moderation, I
> never know how long it will take to appear on the list.
>
> ====================================================
>
> On Mon, 15 Nov 2010 13:35:53 -0800
> Strumpets Delight <strumpetsdelight@...> wrote:
>
> > Do you have documentation of these claims Eric?
>
> The best source I know about what can happen from too much iron
> is an aritcle in the CDC's Emerging Infectious Diseases journal
> a few years ago. It does a very good summary.
>
> ----------------------------------------------------
>
> From http://www.cdc.gov/ncidod/EID/vol5no3/weinberg.htm:
>
> Iron Loading and Disease Surveillance
> Eugene D. Weinberg
> Indiana University, Bloomington, Indiana, USA
>
> ...
>
> Excessive iron in specific tissues (iron loading) promotes
> infection, neoplasia, cardiomyopathy, arthropathy, and a
> profusion of endocrine and possibly neurodegenerative
> disorders.
>
> ...
>
> ... Moreover, in infectious diseases, inflammatory
> diseases, and illnesses that involve ischemia and
> reperfusion, iron causes reactions that produce superoxide
> radicals. ...
>
> ... To survive and replicate in hosts, microbial pathogens
> must acquire host iron. Highly virulent strains possess
> exceptionally powerful mechanisms for obtaining host iron
> from healthy hosts. In persons whose tissues and cells
> contain excessive iron, pathogens can much more readily
> procure iron from molecules of transferrin that are
> elevated in iron saturation. In such cases, even microbial
> strains that are not ordinarily dangerous can cause
> illness. Markedly invasive neoplastic cell strains can
> glean host iron more easily than less malignant strains or
> normal host cells. Moreover, iron-loaded tissues are
> especially susceptible to growth of malignant cells.
>
> ...
>
> Table 1. Iron loading in specific tissues and increased
> risk for disease
>
> Tissue type    Disease
> ----------------------- ---------------------------------
> Alveolar macrophages    Pulmonary neoplasia and
>             infection
>
> Anterior pituitary    Gonadal and growth dysfunction
>
> Aorta; carotid and Atherosclerosis
>             coronary arteries
>
> Colorectal mucosa    Adenoma, carcinoma
>
> Heart    Arrhythmia, cardiomyopathy
>
> Infant intestine    Botulism, salmonellosis, sudden
>             death
>
> Joints    Arthropathy
>
> Liver    Viral hepatitis, cirrhosis,
>             carcinoma
>
> Macrophages    Intracellular infections
>
> Pancreas    Acinar and beta cell necrosis,
>             carcinoma
>
> Plasma and lymph    Extracellular infections
>
> Skeletal system    Osteoporosis
>
> Skin    Leprosy, melanoma
>
> Soft tissue    Sarcoma
>
> Substantia nigra    Parkinson's disease
>
> ...
>
> Table 2. Microbial genera with strains whose growth in body
> fluids, cells, tissues, and intact vertebrate hosts is
> stimulated by excess iron
>
> ...
>
> Excessive iron is correlated with synovial damage in
> rheumatoid arthritis and with impaired glucose metabolism
> in diabetes. The association of Chlamydia pneumoniae and
> excessive iron with cardiovascular disease is well
> established. Growth of this pathogen is strongly suppressed
> by iron restriction.
>
> ----------------------------------------------------------
>
> Take a look at table 2. As much as I'd like to put it
> here, trying to format it to fit is pretty tough.
>
> ----------------------------------------------------------
>
> Now keep in mind that much of this is from too much iron. The
> problem is that there is no magical line between too much and
> not too much. So just a little excessive iron that may affect
> one condition might not have much affect on other conditions.
>
> Some other sources on the Internet you might want to read
> include:
>
> http://articles.latimes.com/1992-09-09/news/mn-156_1_heart-attacks
>
>     According to the report, men with high amounts of iron
>     in their bodies had twice as much risk of heart attacks
>     as men with lower amounts of iron--after known heart
>     disease risk factors, such as cholesterol levels and
>     smoking, were taken into account.
>
>     ...
>
>     A decade ago, Sullivan theorized that women are
>     protected against heart attacks until after menopause
>     because of the large amounts of iron they lose each
>     month in menstrual blood. Despite the exclusion of
>     women from the Finnish study, Sullivan called the
>     findings "especially relevant" to his theory.
>
>     The Finnish researchers, led by Dr. Jukka T. Salonen
>     and Riitta Salonen said their findings raise the
>     question of whether current dietary recommendations for
>     iron intake should be revised for men and possibly for
>     post-menopausal women.
>
>     ...
>
>     Sullivan suggested that, based on the findings, the
>     level of iron considered normal may need to be lowered.
>
> http://www.straightdope.com/columns/read/2548/does-giving-blood-reduce-your-chances-of-getting-heart-disease
>
>     The iron hypothesis arose from the observation that
>     premenopausal women have a much lower incidence of heart
>     disease than men but that after menopause the difference
>     narrows dramatically. Originally it was thought this had
>     something to do with sex hormones. However, men given
>     estrogen suffer more heart disease than normal, and
>     women who stop menstruating due to surgery but still
>     have estrogen-producing ovaries suffer increased heart
>     problems nonetheless.
>
>     In 1981, trying to make sense out of this, pathologist
>     Jerome Sullivan proposed an alternative explanation:
>     "The greater incidence of heart diseases in men and
>     postmenopausal women is due to higher levels of stored
>     iron in these two groups." Evidence: (1) diseases that
>     cause iron buildup often result in heart failure; (2)
>     the older a man gets, the more iron he accumulates; (3)
>     after menopause, stored iron in women rises to the
>     level found in men; and (4) the types of heart disease
>     found in affluent countries are rare among impoverished
>     peoples with iron-deficient diets. Sullivan's
>     recommended treatment: "regular phlebotomy," medicalese
>     for having someone extract your blood.
>
>     Some later research supported Sullivan's theory. The
>     most recent study I've seen, published in Heart by
>     David G. Meyers et al, examined 3,900 male participants
>     in the Nebraska Diet Heart Study and found that
>     nonsmokers who had donated blood in the past three
>     years had a 30 percent lower risk of major heart
>     problems. (Curiously, donating multiple times during
>     the three years, as opposed to once or twice, brought no
>     additional benefit.) Other researchers conjectured that
>     iron interacts with LDL (bad) cholesterol to promote
>     atherosclerosis, and there's a lot of stuff about free
>     radicals and so on that I don't have the heart to
>     explain.
>
> http://www.ncbi.nlm.nih.gov/pubmed/11087066
>
>     Iron is a vital element in life. However, it may
>     participate in diverse pathological processes by
>     catalyzing the formation of reactive oxygen free
>     radicals. During the past decade, considerable evidence
>     has supported the role of oxidative stress in the
>     development of atherosclerosis and related
>     cardiovascular diseases. The oxidation of low-density
>     lipoprotein (LDL) and lipid is believed to be one of the
>     crucial events leading to plaque formation in
>     vasculature. It has been hypothesized that iron-mediated
>     oxidation is involved in this process. In favor of this
>     idea, several epidemiological studies have shown that
>     the level of body iron stores is positively correlated
>     with the incidence of coronary heart disease in human
>     populations. However, some studies have yielded
>     conflicting results. Recently, studies conducted in our
>     laboratory and others have demonstrated that iron
>     deposition is prominent in human atherosclerotic
>     lesions. The iron deposits appear to colocalize with
>     ceroid, which is an end product of extensively oxidized
>     lipid and protein complex, in lesions, providing
>     histological evidence to support the iron hypothesis.
>     Additional experiments in animals have further revealed
>     that the severity of atherosclerosis can be markedly
>     influenced by iron overload or deficiency.
>     Collectively, these data provide a strong pathological
>     basis to support the detrimental role of iron in
>     vascular damage and progression of the disease.
>
> http://www.ncbi.nlm.nih.gov/pubmed/10421276
>
>     Strong epidemiological evidence is available that iron
>     is an important factor in the process of
>     atherosclerosis. Epidemiological studies, eg,
>     prospective follow-up studies in blood donors, may
>     clarify the cardiovascular benefits of iron depletion.
>     Knowledge of the molecular mechanism of iron-related
>     cardiovascular disease is still limited.
>
> http://www.jvascnurs.net/article/S1062-0303(00)38179-1/abstract
>
>     This article summarizes research conducted during the
>     last 2 decades that addresses the idea that stored iron
>     plays a role in the pathogenesis of atherosclerosis and
>     that iron reduction through phlebotomy may play a role
>     in the treatment or prevention of atherosclerosis. Body
>     iron stores rise after adolescence in men and menopause
>     in women. This rise has been linked to the pathogenesis
>     of atherosclerosis through iron-induced oxidation of
>     low-density lipids and foam cell formation. However, the
>     available evidence on the iron hypothesis remains
>     circumstantial. Reduction of body iron stores in the
>     setting of a controlled, prospective intervention trial
>     is necessary to determine whether the amount of stored
>     iron is related to clinically meaningful vascular
>     disease.
>
> -----------------
>
> From what I read once, but don't remember precisely here,
> excess iron is necessary for the formation of atheroclerosis --
> no excess iron, litte or no atheroclerosis.
>
> Eric
>
>
> [Non-text portions of this message have been removed]
>